A homeopathic preparation diluted so far that no arsenic is expected to remain. Human evidence is thin and conflicting, most consistent with no effect beyond a dummy treatment. The real danger lies in poorly made products that still contain arsenic, which have caused serious liver injury, and in skipping proven care. (Full Review)
| Marker | Target | Why |
|---|---|---|
| ALT (alanine aminotransferase) | ≤ 25 U/L (men), ≤ 22 U/L (women) | Detects liver-cell injury, the documented serious harm |
| AST (aspartate aminotransferase) | ≤ 26 U/L | Confirms and contextualizes liver injury alongside ALT |
| Total bilirubin | 0.3–1.0 mg/dL | Rising levels signal impaired liver clearance |
| eGFR (estimated glomerular filtration rate) | ≥ 90 mL/min/1.73 m² | Kidney function governs arsenic excretion |
| Urinary total/speciated arsenic | Below laboratory detection | Direct check for residual arsenic exposure from the product |
Cadence: Recheck liver enzymes and kidney function at roughly 4 weeks after starting, then every 3–6 months while use continues, with prompt unscheduled testing whenever symptoms suggesting toxicity appear. A single short course of a verified product generally needs only symptom-triggered testing.