GHRP-2 for Health & Longevity - Quick Reference Sheet

GHRP-2 for Health & Longevity

Created on 08/09/2026 – Quick Reference based on Evidence Review created using AI4L / Opus 5 Audit

An injected peptide that prompts the pituitary gland to release its own growth hormone in bursts. Best-documented effects: growth hormone and liver growth factor stay raised for at least a month, and hunger rises sharply. Japan licensed it only as a one-time pituitary test. No study of muscle, fat, bone, strength, or long-term outcome in healthy adults exists. (Full Review)

Protocol

Dose
100 µg
Subcutaneous into abdominal fat with a 29–31 gauge insulin syringe; roughly 1 µg/kg
Frequency
1–3× daily
Fasted; split dosing is standard rather than one large dose, and the ceiling is one agonist
Timing
Bedtime
Further slots on waking and 30–60 min after resistance training; not within two hours after a meal
Time to effect
Growth hormone
15–30 min
Peak after the first injection, returning toward baseline by 60–120 min
IGF-1
24 hours
Elevated plateau reached and held through 30 days of continuous dosing
Body composition
Months
Twelve months in the one long trial of a same-receptor agonist, without strength gain

Benefits

Contraindications
  • Active or recently treated malignancy (within 5 years, excluding basal cell skin cancer)
  • Diabetic retinopathy or proliferative retinal disease
  • Untreated prolactinoma or prolactin above the laboratory reference range
  • Acromegaly or any growth hormone-secreting tumour
  • Pregnancy and lactation
  • Under 18 with open growth plates (outside specialist paediatric endocrine supervision)
  • Untreated adrenal insufficiency
  • New York Heart Association Class III–IV heart failure
  • Estimated glomerular filtration rate below 30 mL/min/1.73 m²
  • Uncontrolled type 2 diabetes (HbA1c above 8.0%)
  • Athletes subject to World Anti-Doping Agency testing
Key Interactions
  • Insulin and insulin secretagogues (glimepiride, glipizide, glyburide, injected insulin)
  • Glucocorticoids (prednisone, dexamethasone, hydrocortisone)
  • Thyroid hormone, oestrogen, and testosterone (oral oestrogen in particular)
  • Somatostatin analogues (octreotide, lanreotide)
  • Dopamine antagonists (risperidone, haloperidol, amisulpride, metoclopramide) and agonists (cabergoline, bromocriptine)
  • Other growth hormone axis agents (sermorelin, tesamorelin, CJC-1295, ipamorelin, GHRP-6, hexarelin, ibutamoren, anamorelin, recombinant human growth hormone)
  • Non-steroidal anti-inflammatory drugs (ibuprofen, naproxen)
  • Supplements with additive growth hormone effect (L-Arginine, L-Ornithine, L-Lysine, L-Citrulline, glycine)
  • Carbohydrate or protein within roughly two hours of dosing; high-dose niacin

Risk & Side Effects

  • High: Increased appetite and weight gain; activation of the cortisol axis; prolactin elevation; reduced insulin sensitivity and rising fasting glucose; prohibited substance status in tested sport; product adulteration and unverified content
  • Medium: Fluid retention, joint pain, and carpal tunnel syndrome; partial loss of response with continuous exposure; injection-site reactions and acute autonomic symptoms
  • Low: Elevated IGF-1 and cancer-related concern; cardiovascular and blood pressure effects
  • Speculative: Countering the longevity signal by sustaining growth signalling; rebound suppression of the growth hormone axis after discontinuation; gonadal suppression from sustained prolactin elevation

Monitoring

Marker Target Why
IGF-1 Upper-middle third of the age- and sex-adjusted range; typically 150–250 ng/mL at 40–65 The single integrated readout of whether the protocol is doing anything
Fasting glucose 75–86 mg/dL (4.2–4.8 mmol/L) Detects the compound's principal metabolic liability early
Fasting insulin 2–5 µIU/mL More sensitive than glucose to the loss of insulin sensitivity that precedes any glucose change
HbA1c 4.8–5.3% Confirms whether short-term glucose changes have become a sustained shift
Prolactin Men below 10 ng/mL; non-pregnant women below 15 ng/mL GHRP-2 raises prolactin at every effective dose, and sustained elevation suppresses gonadal function
Morning cortisol 10–15 µg/dL drawn between 07:00 and 09:00 The peptide drives the cortisol axis at a magnitude comparable to a direct corticotropin-releasing hormone challenge
Fasting lipid panel Triglycerides below 80 mg/dL; HDL cholesterol above 55 mg/dL; LDL cholesterol interpreted alongside apolipoprotein B Growth hormone alters fat breakdown and lipoprotein handling
Comprehensive metabolic panel with liver enzymes ALT and AST both below 25 U/L General safety screen, and it captures sodium and potassium relevant to fluid retention
Thyroid panel (TSH, free T4, free T3) TSH 0.5–2.0 mIU/L; free T3 in the upper third of range Thyroid status modulates growth hormone output, so a blunted response can reflect untreated thyroid disease
PSA, men over 45 Below 1.0 ng/mL, with the year-on-year rate of change more informative than the absolute value Prudent surveillance given sustained IGF-1 elevation

Cadence: Full fasting panel at baseline before the first dose; fasting glucose, fasting insulin, and IGF-1 repeated at 6 weeks; HbA1c, prolactin, and morning cortisol added at 12 weeks; full panel then every 3–6 months for as long as dosing continues, plus once at each off-cycle break. Blood pressure and body weight weekly at home.

Qualitative Assessment

  • Sleep depth and morning restoration: whether waking is easier and sleep feels less fragmented
  • Appetite and eating control: the direction and size of hunger change, and whether meal structure is holding
  • Recovery between training sessions: perceived soreness duration and readiness to train
  • Joint comfort and hand symptoms: new nocturnal wrist numbness, tingling, morning hand stiffness, or ring and shoe tightness
  • Libido and sexual function: a decline is the most accessible early indicator of prolactin-mediated gonadal suppression
  • Energy, mood, and cognitive clarity: daytime energy stability and subjective sharpness